GERONTOLOGY SYMPOSIUM 27 May 2017

A chance to meet us in person and get the latest update in Age Management:

GERONTOLOGY SYMPOSIUM 27 May 2017

27. May 2017

Grand Hotel Kempinski Geneva

Life Sciences, Health Care & Medical

www.gerontology2017.com

Experts’ opinion on current approaches in anti-ageing medicine and gerontology.
Symposium will take place on May 27, 2017 in Geneva with the focus on expert opinion on current trends and tendencies in gerontology targeted to inhibit too fast processes of ageing and bring them within physiological limits estimated as such in modern gerontology and anti-ageing medicine. Evidence-based data, safety and scientific argumentation will be given priority.

World renowned experts have been invited to draw up recommendations for application of carefully argued means and methods in medical practice.

WHERE?
Grand Hotel Kempinski Geneva

19, Quai du Mont-Blanc
1201 Geneva

Phone + 41 22 908 91 81
Phone + 41 22 908 90 91

group.grandhotelgeneva@kempinski.com
www.kempinski.com/en/geneva/

 

Aging

ALL ABOUT METABOLISM
If there’s one thing that bonds human beings together, it’s probably the fact that we all age, whether we like it or not. It’s a big deal for us, as we are the only known species on Earth to celebrate birthdays, right?

While some of us embrace aging (and wrinkles and white hair and baldness), others are more concerned on how to avoid aging.

If you’re part of the latter group, you may be interested in reading a study published in the journal Cell, where scientists explain why humans get old and discuss strategies to improve longevity.http://www.cell.com/cell/pdf/S0092-8674(16)30981-3.pdf
According to the scientists, aging, as well as development and maturity, is ultimately determined by metabolism — the process by which our body converts what we eat to energy. Basically, as we age, our metabolism becomes less efficient, which in turn, damages our DNA and causes our cells to stop functioning efficiently — which can be detrimental to our health.

ANTI-AGING INTERVENTIONS
True, aging is inevitable and there seems to be no plausible way to absolutely stop it, much less reverse it; but because metabolism is likewise linked to nutrition, there are ways in which we can at least slow down the process.

Scientists identified what they call “Westernized lifestyle” as an aging accelerator. It’s characterized by hypercaloric nutrition with excess fat and protein intake but limited amount of healthy food, exposure to environmental toxicants, and exaggerated sedentariness — things that are highly discouraged if you want to live long.

So if you want to increase your health span, scientists recommend that you follow these metabolic interventions:

  1. Carbohydrate metabolism: Suprefort, Endoluten, Ventfort
  2. Lipoprotein metabolism: Svetinorm, Suprefort, Ventfort
  3. Mediterranean diet. It’s composed of healthy fats like olive oil, vegetables, whole grains, nuts and fish, and little red meat or sugar.
  4. Caloric restriction. It means reduced intake of calories but without causing malnutrition. Use the DNA-Nutrition Test from Novogenia.(www.dna-sport.eu) 5.Well, of course it’s on the list. We all know how important physical fitness is to longevity.

What is a stroke? | Circulatory System and Disease, can I prevent and repair a stroke?

OLD DRUG, NEW TREATMENT
Researchers from the University of Manchester have developed a new treatment that could limit the damage caused by strokes and also promote repair in the affected area of the brain. What’s more, the drug they’re using has already been clinically approved.

The researchers’ study is published in Brain, Behavior and Immunity, and it recounts how they developed their treatment using mice bred to develop ischemic strokes, the most prevalent type of stroke and one that occurs when an artery that supplies oxygen-rich blood to the brain is blocked. Soon after the mice experienced a stroke, the researchers treated them with interleukin-1 receptor antagonist (IL-1Ra), an anti-inflammatory drug that is already licensed for use in treating rheumatoid arthritis.

They noticed a reduction in the amount of brain damage typically observed after a stroke and also noted that the drug boosted neurogenesis (the birth of new cells) in the areas that did experience brain damage in the days following the treatment. The mice even regained the motor skills they lost due to the stroke.

CERLUTEN – is a natural non-hormonal bioregulator
obtained from the brain tissue of young animals and is used to restore and maintain the central nervous system and the brain. Its structure consists of the complex of amino acids which influence directly on the DNA structure of the brian, replace damaged or aged molecules and restore the protein synthesis, thus providing optimal functionality of the brain. Cerluten is used to treat and prevent all brain diseases and dysfunctions such as consequences of apoplectic stroke, craniocerebral injury, encephalopathy caused by ischemic аtherosclerosis. Prevents the brain from age-related alterations.

INDICATION FOR USE

Atherosclerosis
After Stroke Therapy
Memory Loss and Lack of Concentration
Alzheimer’s and Parkinson’s Diseases
Multiple Sclerosis
Chronic Fatigue Syndrome
Neuralgia and Peripheral Neuropathy

DOSAGE

Active substance: peptide complex A-5 (peptides from the brain tissue)
Excipients: microcrystalline cellulose (E460), beetroot sugar, lactose, starch, Tween-80
Recommendations for use for adults: 1-2 capsules 1-2 times a day with meals. Duration: 1 month. Repeat every 3-6 months.

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Does Reducing Stress Impact Telomere Length?

Several studies link telomere length and telomerase activity with stress reduction activities and suggest a mechanism for the health benefits associated with lower stress such as improved sleep, better immune function and a more positive mental outlook.

So you’re stressed. You are not alone – the American Psychological Association’s 2015 Stress in America Survey reports that 78% of American adults reported having experienced at least one symptom of stress last year. There is a considerable body of research that associates chronic stress exposure with telomere length.

Research on whether and how lifestyle change interventions that aim to reduce stress also change telomere length is still in its early stage. Below, I have summarized some of the various pilot studies that address this question. In three of the below studies, the authors measured telomerase activity as a potential mechanism an indirect proxy for telomere growth. It therefore warrants a brief overview of the functional role of the enzyme telomerase in the growth and maintenance of telomeres.

Telomerase activity is crucial for preservation of telomeres’ function and structure. Technically speaking, telomerase is an enzyme made of protein and RNA subunits that that restores telomeres by adding TTAGGG telomeric DNA sequences when they are shortened due to mitosis, or cell division or other reasons. When the cell’s telomeres become critically short, the cell becomes senescent and cell division stops. Telomerase replaces the bit of DNA lost in each cell division, allowing the cell line to divide without ever reaching the limit. The below studies that look at stress reduction activities and their impact on telomeres use both telomere length measurement and telomerase activity levels to assess association.

In a paper published in 2008 by Dean Ornish and others in Lancet Oncology, 30 men with biopsy-diagnosed low-risk prostate cancer were recruited to participate in a comprehensive lifestyle change program. The program included a low fat, whole foods, plant-based diet high in fruits, vegetables, unrefined grains, legumes, and low in refined carbohydrates; moderate aerobic exercise, stress management (gentle yoga-based stretching, breathing, meditation, imagery, and progressive relaxation techniques), and group support sessions. Telomerase activity was measured at the baseline before the start of program and after 3 month from peripheral blood mononuclear cells (PBMCs). There was a statistical significant increase in telomerase activity after 3 month of comprehensive life style change. More importantly, increase in telomerase activity is associated with a decrease in lipoprotein (LDL) cholesterol and psychological distress.

In a follow-up paper by the same authors, PBMC telomere length was measured from the baseline (before the start of the lifestyle change program) and 5 years later in ten men from the 2008 study cohort.  After the three months program, the participants could meet on their own for 2-4 hour meetings per month for the duration of the study with a physician or nurse on site. The control group consists of 25 men with low-burden prostate cancer who had chosen active surveillance rather than conventional treatment (for reasons unrelated to the study). Telomere length was measured with the qPCR method. Average telomere length increased from baseline to 5 years in the lifestyle intervention group, but decreased in the control group as expected. Adherence to lifestyle changes was positively associated with telomere length after adjustment for age and the length of follow-up. This is the first intervention study that has shown a significant change in average telomere length when compared with a non-intervention group. I should point out that this is a non-randomized, pilot study with a very small sample size, and the comparison group came from a different study (although with the same inclusion, exclusion criteria). So larger size, randomized studies are needed to test if the results can be replicated.

Meditation is widely accepted as a way to reduce psychological stress and enhance well-being. However, the mechanistic links between meditation and its psychological and physical benefits are poorly understood. Recent studies suggest telomere maintenance maybe one of the cellular systems that is impacted by meditation. In the journal Psychoneuroendocrinology, Jacobs et al reported the effects of a three-month intensive meditation retreat on telomerase activity.

The participants were sixty men and women (aged 21-69) recruited to study the relation between meditation and well-being. They were matched on demographic variables (sex, age) and meditation and randomly assigned to either an on-site, three month meditation retreat or a wait-list control group. The meditation retreat was instructed by Alan Wallace, Ph.D., a well-known Buddhist scholar and practitioner. The meditation focuses on the cultivation of attentional skills and the generation of benevolent mental states. Telomerase activity was measured from PBMCS after the 3-month retreat in the participants and in the control group who were on the waiting list and were about to start the retreat. Psychological assessments included mindfulness, purpose in life and perceived control and neuroticism, measured at baseline and after 3 months. Mindfulness was surveyed by the 37-item Five Facet Mindfulness Questionnaire (FFMQ), which assesses five facets of Mindfulness (observing or noticing experience; acting with attentional awareness or avoiding automatic pilot; non-reactivity to internal experience; describing or labeling feelings; nonjudging of experience). Purpose in Life was measured by a 9-item questionnaire as part of the Well-Being Scale designed by Dr. Carol Ryff that assesses changes in a person’s sense that life is meaningful, organized around clear aims, and clearly directed. Perceived Control was measured by the 9-item Environmental Mastery subscale within the Well-Being Scale to measure perceived control over situations and circumstances.

As expected, retreat participants scored higher on the mindfulness, purpose in life and perceived control questionnaires and lower on the neuroticism score after 3 months. Telomerase activity is higher in the retreat group compared to the waiting-list control group. Using a statistical tool called mediation analysis, the authors showed that the group difference in telomerase activity can be explained by changes in Perceived Control, Neuroticism, and Purpose in Life suggesting that the intensive meditation program affects telomerase activity through improvement in Perceived Control, Neuroticism, and Purpose in Life scores.

In another study by the same group of authors, participants were enrolled in a one-month Insight meditation retreat, a mindful-based mediation program in the Buddhism tradition. Telomere length was measured in PBMCs at the beginning of, and three weeks into the retreat in 26 participants. The control group has 30 age-, gender-, and meditation-experience matched participants who also provided blood at baseline visit and 3 weeks after. While the two groups did not differ at baseline; the retreat group’s telomeres were significantly longer at three weeks than at baseline, whereas the control group showed no change. This pilot work provides the first evidence that a short, but intensive mediation program is associated with telomere lengthening.

The control group was asked to relax in a quiet place with their eyes closed while listening to instrumental music on the relaxation CD for 12 minutes every day at the same time for eight weeks. Telomerase activity was measured in PBMCs at baseline and after 8 weeks in both groups. A battery of psychological assessments was also administered. The Mental Health composite score (MCS score) assessed health-related quality-of-life, mental, physical, and social functioning. The Hamilton Rating Scale for Depression was used to quantify mood symptoms. The Mini-Mental State Examination (MMSE) was used to measure cognitive performances. The yogic practice group had improved Mental Health Summary (MCS) score and cognitive function and decreased depression score. The meditation group had increased telomerase activity, compared to no change in the control group. More importantly, the increased telomerase activity was correlated with a decrease in levels of depression and increase in mental health score when both groups are combined. This suggests that stress reduction by meditation is accompanied by an increase in telomerase activity.

So far, all three studies I talked about in this blog showed an increased telomerase activity with lifestyle change. However, recent studies have also shown that the relationship between telomerase activity and disease risk factors maybe U-shaped where both low and high telomerase activity are associated with risk factors. Specifically, the combination of high telomerase activity and short telomere length is associated with low socioeconomic status, clinical depression (MDD) and coronary artery calcification.

Initial research on telomeres and stress reduction techniques seems be consistent with other research that points to the health benefits of stress reduction techniques such as improved mood, sleep and immune function. The above studies are compelling in that they suggest that telomere preservation and maintenance is a significant mechanism that results in the positive physical and psychological outcomes associated with meditation and stress reduction measures.

As aging is associated with a decline in the synthesis of protein, it is logical to assume that if the synthesis is restored aging can be slowed down. As it turns out this assumption is correct. Scientists V. Khavinson and V. Morozov have found a way to repair protein production and have achieved incredible results. Using the following methods can increase lifespan by 20-40%.

Triggering molecules
The information about different proteins is stored in the DNA. In order to launch protein synthesis a DNA fragment, a gene, has to be activated by triggering molecules, peptides. Peptides are relatively short chains of amino acids and they are an essential part of the cell self-regulating mechanism:
Cells constantly degrade aged proteins by breaking them down into amino acids and peptides. Some of these peptides match specific parts in the cell DNA following the lock-key principle. As a result, the peptide resumes the synthesis of the protein from which it was originally built. When the protein ages, it is fragmented into the same peptides once again. All of this forms a circular process that is vital to cell life.

Epitalon and Endoluten are peptides which were originally developed based upon the action of epithalamin, a hormone produced by the pineal gland. This hormone was found to stimulate the production of telomerase, an enzyme which plays a role in maintaining telomere length. Telomeres are non-coding terminal regions of DNA strands which preserve the integrity of the strand. With each revision, telomeres are shortened until the DNA strand cannot be further replicated. This process is highly implicated in the ageing process. Elongating telomeres theoretically extend the lifespan of a copy of DNA and allows it to replicate more times than usual. This was the theory behind the development of Epitalon®, a synthetic version of epithalamin which also stimulates the production of telomerase. Indeed, this theory has been confirmed in vitro inhuman cell cultures.

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FIRST FACE MASK TO REJUVENATE THE SKIN CELLS

Aesthetic medicine – is medicine of beauty and youth
Main target for aesthetic medicine is to slow down the processes of skin ageing.
For the first time new technologies have been developed in order to postpone and
slow down the processes of skin ageing.
The problem that was not possible to resolve with aesthetic therapy is now solvable
due to the innovational mediсines – Khavinson peptides®

How can we protect our skin from ageing to the maximum?
Unique peptide geroprotectors are scientifically proven to slow down the
processes of ageing
One of them is EPITALON – first peptide to control the processes of
ageing.

NANOPEP mask eng (2)

 

The major cause of death today can be linked to inflammation

Inflammation and the body’s inflammatory response
Everybody has heard about the term inflammation, but many people are not quite sure what it means and why we have it. Inflammation is a response that your body mounts. Through the process of evolution, we’ve developed the ability to mount this response to deal with the things that used to kill us.Up to about 100 years ago, the common killers were infection and trauma.

Whenever we have an infection in our body, or we have trauma and tissue damage, our immune system has evolved the ability to mount an inflammatory response to fix the problem so that we can live. So what happens with an inflammatory response whenever we have localized trauma or infection, the immune system releases chemicals which cause the blood vessels in the area to dilate so more blood can go to the area. That is why you see increased redness and feel increased warmth in the area.

The blood vessels get leaky so that cells that are coming in on that increased blood supply can get out of the blood vessels and fluid leaks out of the blood vessels at the same time, which is why the area swells up. The cells go around and there are macrophages that eat the bacteria or a virus causing the infection that eats the damaged tissue from the trauma. Then there are other cells such as fibroblasts that come in and repair the damage that is done and then once it’s all fixed the inflammatory response settles down, and the swelling and the redness and the heat and the pain all goes away. So that’s an acute, localized, temporary response to the common things that used to kill us, infection and trauma.In the last 100 years, we’ve controlled infection and trauma quite well. Trauma is not a big problem anymore. We don’t have tigers and dinosaurs, and we don’t have to go hunting for food, and we don’t have people with clubs and spears (inaudible) our cave, and we have occupational health and safety rules, and helmets and seat belts, so trauma is a relatively minor problem now.

For instance, heart attacks and strokes, cardiovascular diseases as a group, are the largest killer in western society, and they are all caused by chronic, low-grade, inflammatory damage to the blood vessels and this damage that’s happening via the blood vessels over decades. You don’t feel that happening. You don’t feel that until that blood vessel bursts or blocks, and you have your heart attack and stroke.

Similarly with cancers, a normal cell in your body doesn’t suddenly turn into cancer cell overnight. You have got to spend decades damaging the DNA and the epigenome switches around the DNA until that one particular cell now can reproduce itself in an uncontrollable manner, then you have your cancer. So there’re decades of damage done to the cell before it turns into a cancer cell. Dementia, which is going to be one of the biggest killers in 40 years, it’s going to be one of the biggest, if not the biggest killer. When you are finished growing, you have about 86 billion brain cells, and we lose about 8,000 a day.Now you can lose more than that, you can lose less than that, but if you have an accelerated rate of brain cell loss over your life, eventually, your brain gets so small and so less cognitive processing capacity that you can’t look after yourself anymore and at that stage we say you are a person with dementia, and 50% of 85-year-old have dementia, but that didn’t happen between 84 and 85 years old, that was a 50 year process of going from 84 billion brain cells down to not enough to look after yourself anymore, and that’s all caused by neuro inflammation or inflammation in the brain that’s affecting brain cell health, how well I work, and killing off the brain cells and so on and so on for the other chronic degenerative diseases.

80-90% of people in the west now die of chronic degenerative diseases caused by chronic low-grade inflammation. The trick to having a long healthy life is to minimize the amount of inflammation that’s in your body over your lifespan. You do that two ways: you remove the triggers that are triggering off this inflammatory response that is supposed only to be there occasionally to deal with acute trauma and infections, and you dampen down whatever inflammation is there.

There are thousands of potential triggers that we are all exposed to, some more than others, depending on your lifestyle primarily and environmental habits. Potential triggers that set off inflammatory response is not enough sleep, too much sleep, not enough exercise, too much exercise, too much energy in your diet, not enough nutrition in your diet, too many chemicals in your air, your water, and your food.The immune system triggers off the inflammatory response whenever it identifies foreign chemicals such as:
Insecticides
Herbicides
Pesticides
Fungicides
Colourings
Flavourings
Preservatives
Sweeteners
Heavy metals
Antibiotics

The immune system sees foreign chemicals  as an infection, foreign invader or source of physical damage and trauma that needs repairing. Too much stress, not enough stress, all these lifestyle factors both how we live our life and where we live our life, these are the things that determine how much people die.Most cancers, 80-90% are environmental, they are not genetic. Some people have genetic risk factors for different cancers, but 80% of cancers are not caused by inherited factors, they are caused by environmental factors, lifestyle factors that are based on how people live their lives. 80-90% of cancers are caused by environmental factors; these are all decisions we make through our life and so there are things that we do that we shouldn’t do or there are things we should do that we don’t do that protects us from the damage and inflammation that causes these chronic degenerative diseases.

Making decisions and identifying what are your triggers for setting off the inflammatory process in your body and there are triggers that everybody has in common. All the chemicals in the environment are triggers for everybody, but you might have particular food intolerance that set off the inflammatory process in you, or you might have intolerance for certain chemicals or heavy metals, because you have inherited genes that are not so effective at removing them from your body so they accumulate in your body more readily. There are specific things that are sensitive and individualized, and there are things that affect everybody. You want to try to identify and remove all those triggers and then you want to also do some anti-inflammatory steps to minimize whatever inflammation is left over once you have optimized your triggers.

Things you can do to minimize inflammation in your body is to get adequate sleep, getting adequate exercise, not too much energy input, but lots of nutrition, antioxidants, phytonutrients in plants, nuts, seeds, fruits, vegetables. Vitamin D is a strong anti-inflammatory, the omega-3 fatty acids are especially the EPA in fish oil is a strong anti-inflammatory, Curcumin and the Indian spice turmeric is a strong anti-inflammatory, Silymarin is the active ingredient of a herb called milk thistle which has been used for thousands of years for liver support but it’s also a strong anti-inflammatory, especially for the brain. There are many other anti-inflammatory herbal phytonutrients and supplements you can have to support and minimize the amount of inflammation you have in your body over time.

Minimizing the amount of inflammation you have in the body is one of the best things you can do to not only have your body work well now, have your brain work now, your liver, your heart, your muscles, everything work well now so you feel and function optimally but it’s also a very powerful thing to maintain your health over the next decades of your life. Identifying the triggers and removing them and putting in the good stuff. It’s all about taking out the bad stuff and putting in the good stuff as they say. That’s what I think you need to know about inflammation.

That´s why you should use our Peptide Bio Regulator VESUGEN

www.peptide-bioregulator.com

Why buying Peptides-Bio Regulators online from an offshore supplier is a gamble with your health.

Why buying peptides online from an offshore supplier is a gamble with your health.
No regulatory control of the peptide product quality, its purity or potency
No regulatory control of the advertising
No accountability for misleading customers
Offshore supply often sells “research grade peptides” yet will not advertise this truth.
When there is no regulatory control, customers must be aware that anything within the website including product certifications of quality may be falsified to mislead potential customers.  Regulatory control protects European consumers. Many of the offshore suppliers will advertise their Research peptides passing them off as Pharmaceutical Grade Peptides.

Research Peptides are not safe for human consumption.
They do not undergo the rigorous protocol required for maintaining the sterility and purity of product.
Research peptides are often found to contain contaminants which are harmful to the body. Since peptides typically get introduced into the body by way of injection, a lack of purity may lead to a patient injecting a plethora of unknown compounds and contaminants directly into the bloodstream.
Overseas suppliers create websites specifically focused on targeting the European peptide market, selling directly to European patients, posing as a local vendor. Be wary of these, as we have just covered, the consumer is not protected when the product comes from an offshore supply.  Without regulatory control, business goes on as usual regardless, and there is no accountability required for misleading customers.

Function of the Thyroid Gland

The thyroid gland is the largest gland in the human neck
It is located at the front of the neck (anterior) under the muscle layers
The thyroid gland is placed in a way that it assimilates the shape of a butterfly. This is visualised as the left and right thyroid lobes (butterfly wings) which wrap around the trachea.
The thyroid gland is located just under the larynx.
The function of the thyroid gland is to regulate the body’s metabolism by taking iodine and converting it into two thyroid hormones called thyroxine (T4) and triiodothyronine (T3).
Thyroid cells are exclusive in that they are the only cells in the body capable of absorbing iodine.
Every cell in the body depends upon thyroid hormones for regulation of their metabolism.
The thyroid gland is under the control of the pituitary gland
More T4 than T3 is released by the thyroid gland, but it is turned into T3 by tissues in the body, using zinc and selenium-dependent enzymes.

Triiodothyronine (T3)
Vital roles in the regulation of the body’s metabolic rate, and oxygen usage of most cells in the body. T3 regulates heart rate, muscle strength, digestion, brain development and bone maintenance. T3 levels are decreased by inadequate dietary iodine / selenium / zinc, by auto-immune damage to the thyroid gland or by obesity / recurrent dieting / mental or physical stress / inflammation (cause T4 to be converted to reverse T3 instead of T3). Increased Reverse T3 blocks and inactivates the T3 receptor.
Thyroxine (T4)
Thyroxine’s vital role is in heart function, metabolism, digestion, brain development, bone health and muscular strength. In the bloodstream thyroxine is converted into an active form of triiodothyronine.
Regulated by the Hypothalamus and Pituitary Gland

TSH
TSH (Thyroid-stimulating hormone) plays a part in the production of thyroxine and triiodthyronine by stimulating the thyroid gland.
Thyroid Gland Disease and Hormone Deficiency

Thyroid disease is fivefold more common in women, and up to 20% of 60+ yo women have some degree of lowered T3 levels (hypothyroidism).
Symptoms of hypothyroidism include fatigue, weakness, un-refreshing sleep, low body temperature, cold sensitivity, weight gain which can’t be lost, decreased intestinal movement (indigestion, nausea, vomiting, constipation), dry/course skin/hair, menstrual irregularities, depression, brain fog, poor memory.
Signs include increased Low Density Lipoprotein / blood pressure / homocysteine / C-Reactive Protein.

 

Thyreogen thyroid extract Peptide Bio Regulator
THYREOGEN is a natural peptide bioregulator extracted from the thyroid gland of young animals. It is used to regulate, restore and protect the function of the endocrine system. It prevents from the dysfunction of the thyroid gland, thus preventing metabolic disorders, hypothyroidism and hyperthyroidism. It works by acting directly to the DNA of the thyroid gland, stimulating repair and regenerative processes, which provide the working optimum level of the whole endocrine system. It is highly effective in preventing autoimmune nervous system disorders in women older 50 years old who enter the menopause period (discomfort, headache, weakness, mental impairment).

INDICATION FOR USE

Metabolic Disorders
Thyroid Diseases
Hyperthyroidism and Hypothyroidism
Thyroid Nodules
Autoimmune Diseases
INSTRUCTION FOR USE

Active substance: peptide complex A-2 (peptides of the thyroid gland)
Excipients: microcrystalline cellulose (E460), beetroot sugar, lactose, starch, Tween-80
Recommendations for use for adults: 1-2 capsules 1-2 times a day with meals. Duration: 1 month. Repeat every 3-6 months.

Bio Regulators “Keeping the Quality of Life” “We work to add life to years and to make active and healthy longevity available to everyone “.

Bio Regulators for:
Digestive system: Svetinorm, Suprefort, Stamakort or Ovagen, Vesugen, Crystagen

Vascular system: Ventfort, Svetinorm, Vladonix or Vesugen, Crystagen

Cardiovascular system: Chelokhart, Ventfort, Svetinorm

Central nervous system: Cerluten, Ventfort, Svetinorm or Pinealon, Vesugen, Ovagen

Immune system: Vladonix, Endoluten, Ventfort or Crystagen, Vesugen, Ovagen

Locomotor apparatus: Sigumir, Ventfort, Vladonix or Cartalax, Vesugen, Crystagen

Carbohydrate metabolism: Suprefort, Endoluten, Ventfort

Respiratory system: Chonluten, Crystagen, Vesugen

Lipoprotein metabolism: Svetinorm, Suprefort, Ventfort

Thyroid gland: Thyreogen, Ventfort

Vision: Visoluten, Cerluten, Ventfort

Kidneys: Pielotax, Ventfort

After radio and chemotherapy, psycho-emotional stress and similar adverse factors: Vladonix, Svetinorm, Endoluten or Vesugen, Ovagen, Chonluten.

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