Videos Archives - The Bioregulator Company

Retinitis Pigmentosa

Maintenance and restoration of functions of the eye, being one of the main sense organs, is among vital issues of modern medicine. 80% of information about environment people get by visual perception. Most of the professional and everyday activities have to do with visual functions, their slackening or loss has the utmost negative effect on the quality of life.
In the structure of the eye retina (nervous tunic of the eyeball) is one of the thinnest layers, being the most complex and highly differentiated tissue. Its complex structure makes it sensitive to light, allowing to perceive color and images and to convert them into a signal being sent straight to the brain. Position of the retina behind other optic structures, direct ingress of sun rays, peculiarities of the blood supply make it vulnerable to both external (sun rays, light striking, radiation) and internal factors. Retina is as a rule damaged in case of the following diseases:  hypertension, diabetes mellitus, renal insufficiency etc. It should be noted that any damage of the retina can lead to the impaired vision or even to complete blindness.
Among the most frequent retinal diseases there are age-related macular degeneration, hereditary retinal degenerations (including retinitis pigmentosa), complicated myopia, diabetic retinopathy. Modern methods of treatment based on the application of the known pharmaceutical substances do not lead to significant results. Usually a prognosis for a disease in these patients is unfavorable (gradual and progressive decrease in the visual functions up to blindness).

For the first time in the world medical and ophthalmological practice an unprecedented technology for restoration and treatment of retinal functions in case of different pathologies (including macular degeneration, retinitis pigmentosa, diabetic retinopathy, thermal injuries, etc.)

has been discovered.



Long Telomeres Protect Against Genetic Predisposition to Age-Related Disease

Long Telomeres Protect Against Genetic Predisposition to Age-Related Disease

Researchers at the Gladstone Institute have shown in a mouse model how longer telomeres can protect carriers of the NOTCH1 haploinsufficiency mutation from developing premature calcification of the aortic valve (Cav). The researchers found that the mice that carried this mutation but had longer telomeres did not develop CAV whereas the mice that had the mutation and shorter telomeres did develop CAV and the degree of telomere shortening followed the severity of disease.

Mice with a genetic mutation for a form of cardiovascular disease that causes premature calcification of the aortic valve (CAV) in addition to longer telomeres, did not develop disease. In contrast, mice with this mutation but short telomeres, did develop CAV and the severity of the disease progression tracked the degree of telomeric shortening.


A chance to meet us in person and get the latest update in Age Management:


27. May 2017

Grand Hotel Kempinski Geneva

Life Sciences, Health Care & Medical

Experts’ opinion on current approaches in anti-ageing medicine and gerontology.
Symposium will take place on May 27, 2017 in Geneva with the focus on expert opinion on current trends and tendencies in gerontology targeted to inhibit too fast processes of ageing and bring them within physiological limits estimated as such in modern gerontology and anti-ageing medicine. Evidence-based data, safety and scientific argumentation will be given priority.

World renowned experts have been invited to draw up recommendations for application of carefully argued means and methods in medical practice.

Grand Hotel Kempinski Geneva

19, Quai du Mont-Blanc
1201 Geneva

Phone + 41 22 908 91 81
Phone + 41 22 908 90 91



If there’s one thing that bonds human beings together, it’s probably the fact that we all age, whether we like it or not. It’s a big deal for us, as we are the only known species on Earth to celebrate birthdays, right?

While some of us embrace aging (and wrinkles and white hair and baldness), others are more concerned on how to avoid aging.

If you’re part of the latter group, you may be interested in reading a study published in the journal Cell, where scientists explain why humans get old and discuss strategies to improve longevity.
According to the scientists, aging, as well as development and maturity, is ultimately determined by metabolism — the process by which our body converts what we eat to energy. Basically, as we age, our metabolism becomes less efficient, which in turn, damages our DNA and causes our cells to stop functioning efficiently — which can be detrimental to our health.

True, aging is inevitable and there seems to be no plausible way to absolutely stop it, much less reverse it; but because metabolism is likewise linked to nutrition, there are ways in which we can at least slow down the process.

Scientists identified what they call “Westernized lifestyle” as an aging accelerator. It’s characterized by hypercaloric nutrition with excess fat and protein intake but limited amount of healthy food, exposure to environmental toxicants, and exaggerated sedentariness — things that are highly discouraged if you want to live long.

So if you want to increase your health span, scientists recommend that you follow these metabolic interventions:

  1. Carbohydrate metabolism: Suprefort, Endoluten, Ventfort
  2. Lipoprotein metabolism: Svetinorm, Suprefort, Ventfort
  3. Mediterranean diet. It’s composed of healthy fats like olive oil, vegetables, whole grains, nuts and fish, and little red meat or sugar.
  4. Caloric restriction. It means reduced intake of calories but without causing malnutrition. Use the DNA-Nutrition Test from Novogenia.( 5.Well, of course it’s on the list. We all know how important physical fitness is to longevity.
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